Differential Diagnosis Classification

Juvenile idiopathic arthritis requires meticulous clinical exclusion of other diseases.

Table 1: Differential Diagnosis Of Childhood Arthritis

Disease CategorySpecific Conditions
Infectious DiseasesSeptic arthritis, viral arthritis (rubella, parvovirus B19, Epstein-Barr virus, hepatitis B), Lyme disease, osteomyelitis, endocarditis.
Reactive And Post-InfectiousAcute rheumatic fever, reactive arthritis (enteric or urogenital triggers), transient synovitis of the hip.
Rheumatic And AutoimmuneSystemic lupus erythematosus, juvenile dermatomyositis, scleroderma, IgA vasculitis, Kawasaki disease, sarcoidosis, mixed connective tissue disease.
Neoplastic DisordersLeukemia, neuroblastoma, lymphoma, bone tumors.
Orthopedic And MechanicalTrauma, growing pains, hypermobility syndromes, Legg-Calve-Perthes disease, slipped capital femoral epiphysis, chondrolysis.
Autoinflammatory SyndromesPeriodic fever syndromes, macrophage activation syndrome, familial Mediterranean fever.
ImmunodeficienciesHypogammaglobulinemia, IgA deficiency, common variable immunodeficiency.
Congenital And MetabolicGout, mucopolysaccharidoses, thyroid disease, scurvy, skeletal dysplasias.

Differentiating Features

  • Acute rheumatic fever causes exquisitely painful, migratory polyarthritis.
  • Malignancy exhibits nocturnal bone pain awakening children from sleep.
  • Malignancy demonstrates disproportionate pain relative to physical findings.
  • Leukemia presents with leukopenia, low-normal platelets, and high erythrocyte sedimentation rate (ESR).
  • Bone marrow examination remains mandatory confirming malignant infiltration.
  • Transient synovitis causes acute hip pain following viral infections.
  • Septic arthritis presents as an acute, painful, erythematous, hot single joint.
  • Synovial fluid aspiration excludes septic arthritis definitively.

Management Principles

Treatment Goals

  • Achieve complete disease remission.
  • Prevent or halt joint damage and deformities.
  • Foster normal psychosocial and physical growth.

Pharmacological Interventions

Table 2: Pharmacotherapy Agents And Indications

Drug ClassSpecific AgentsPediatric DosageClinical Indications And Notes
Non-Steroidal Anti-Inflammatory Drugs (NSAIDs)Naproxen10–20 mg/kg/day PO divided q12h (Max: 1000 mg/day)Initial symptomatic therapy relieving pain and stiffness.
Ibuprofen30–40 mg/kg/day PO divided q6-8h (Max: 2400 mg/day)Initial symptomatic therapy relieving pain and stiffness.
Meloxicam0.125 mg/kg PO once daily (Max: 7.5 mg/day)Initial symptomatic therapy relieving pain and stiffness.
GlucocorticoidsTriamcinolone hexacetonide1 mg/kg per large joint (knee/hip); 0.5 mg/kg per small joint (Max: 40 mg per joint)Intra-articular injection preferred for oligoarthritis.
Prednisolone0.5–2 mg/kg/day PO (Max: 60 mg/day); use lowest effective dose for bridgeSystemic route serves as bridge therapy or controls severe systemic JIA.
Conventional Synthetic DMARDsMethotrexate10–15 mg/m²/week SC or PO once weekly (Max: 25 mg/week)Anchors treatment for polyarthritis and extended oligoarthritis.
Sulfasalazine30–50 mg/kg/day PO divided q12h; start low and titrate upward (Max: 2000 mg/day)Targets enthesitis-related arthritis.
LeflunomideWeight-based: <40 kg: 10 mg every other day or daily; >40 kg: 10–20 mg PO once dailyAlternate conventional DMARD option.
Biologic DMARDs (Anti-TNF)Etanercept0.8 mg/kg/week SC once weekly (Max: 50 mg/week)Targets polyarthritis refractory to methotrexate.
AdalimumabWeight-based: 10 to <15 kg: 10 mg; 15 to <30 kg: 20 mg; ≥30 kg: 40 mg SC every 2 weeksPreferred treating associated uveitis.
Infliximab6 mg/kg IV infusion at weeks 0, 2, 6, then every 8 weeksUsed off-label for refractory polyarticular JIA/uveitis.
Golimumab30 mg/m² SC every 4 weeks (Max: 50 mg per dose)Treats polyarticular JIA.
Biologic DMARDs (Anti-IL-1)Anakinra1–2 mg/kg/day SC once daily (Max: 100 mg/day); up to 4 mg/kg/day in MASHighly effective treating systemic JIA.
CanakinumabWeight-based: ≤75 kg: 4 mg/kg SC every 4 weeks; >75 kg: 300 mg SC every 4 weeksHighly effective treating systemic JIA.
Biologic DMARDs (Anti-IL-6)TocilizumabsJIA: <30 kg: 12 mg/kg IV q2w; ≥30 kg: 8 mg/kg IV q2w
pJIA: <30 kg: 10 mg/kg IV q4w; ≥30 kg: 8 mg/kg IV q4w
Manages systemic JIA and macrophage activation syndrome.
T-Cell ModulatorsAbataceptIV: <75 kg: 10 mg/kg q4w
SC: 10 to <25 kg: 50 mg/week; 25 to <50 kg: 87.5 mg/week; ≥50 kg: 125 mg/week
Inhibits T-cell activation treating polyarticular JIA.
Small Molecules (JAK Inhibitors)TofacitinibOral Solution/Tablet: <40 kg: 5 mg (5 mL) PO q12h; ≥40 kg: 5 mg tablet PO q12hTreats polyarticular JIA targeting intracellular signaling.

Subtype-Specific Approach

  • Oligoarthritis management utilizes NSAIDs or intra-articular corticosteroids initially.
  • Methotrexate addition indicated for extended oligoarthritis or refractory disease.
  • Polyarthritis demands early aggressive therapy utilizing methotrexate.
  • Biologic tumor necrosis factor (TNF) antagonists added upon methotrexate failure.
  • Systemic JIA therapy utilizes corticosteroids alongside interleukin-1 or interleukin-6 antagonists.
  • Enthesitis-related arthritis responds to NSAIDs, sulfasalazine, and TNF inhibitors.
  • Macrophage activation syndrome requires pulse intravenous methylprednisolone and cyclosporine.

Multidisciplinary Rehabilitation

  • Physical therapy prevents fixed flexion contractures and maintains joint mobility.
  • Occupational therapy provides customized splints facilitating activities of daily living.
  • Routine slit-lamp ophthalmologic screening mandatory detecting asymptomatic uveitis.
  • Nutritionist consultation addresses growth failure and osteopenia secondary to chronic inflammation.
  • Psychosocial support essential addressing depression, school absenteeism, and chronic pain impact.