Principles Of Therapy

  • Antiretroviral therapy transforms pediatric HIV from a rapidly fatal disease into a manageable chronic condition.
  • Primary goals include maximal plasma viral load suppression, immune function restoration, morbidity reduction, and normal growth.
  • Combination therapy strictly utilizes at least three drugs from at least two different classes to prevent viral resistance.
  • A universal treat-all policy mandates therapy initiation in all infected children regardless of clinical stage or CD4 count.
  • Early initiation significantly reduces mortality, particularly in infants younger than one year.
  • Treatment success strictly requires adherence exceeding 90% to 95%.

Classes Of Antiretroviral Drugs

Drug ClassMechanism Of ActionKey Examples
Nucleoside Reverse Transcriptase InhibitorsAct as chain terminators during viral DNA synthesis; serve as the dual backbone of first-line regimens.Zidovudine, Lamivudine, Abacavir, Tenofovir.
Non-Nucleoside Reverse Transcriptase InhibitorsBind directly to reverse transcriptase enzyme; possess a low genetic barrier to resistance.Nevirapine, Efavirenz.
Protease InhibitorsInhibit viral protease, preventing virion maturation; exhibit a high genetic barrier to resistance.Lopinavir/ritonavir, Atazanavir/ritonavir.
Integrase Strand Transfer InhibitorsPrevent integration of viral DNA into the host genome; possess a high barrier to resistance.Dolutegravir, Raltegravir.

Regimen selection is strictly stratified by age and weight.

Age And Weight CategoryPreferred Regimen
Neonates (< 4 Weeks)Zidovudine + Lamivudine + Nevirapine (Or Raltegravir).
Children < 6 Years And < 20 kgAbacavir + Lamivudine + Lopinavir/ritonavir.
Children 6 To 10 Years And 20 To 30 kgAbacavir + Lamivudine + Dolutegravir.
Adolescents > 10 Years And > 30 kgTenofovir + Lamivudine + Dolutegravir.

Monitoring Protocol

  • Clinical Monitoring: Assess growth, developmental milestones, adherence, and opportunistic infections at every visit.
  • Viral Load: Targets undetectable levels (<50 copies/mL). Monitored at baseline, 6 months, 12 months, and annually.
  • CD4 Count: Monitored at baseline and every 6 months to guide opportunistic infection prophylaxis.

Treatment Failure Definitions

  • Virological Failure: Plasma viral load remains $\ge$ 1000 copies/mL after at least 6 months of compliant therapy.
  • Immunological Failure: Persistent decline in CD4 count or percentage.
  • Clinical Failure: New or recurrent WHO Stage 3 or 4 events or neuro-regression after 6 months of therapy.

Common Adverse Effects

DrugKey Adverse Effects
ZidovudineAnemia, neutropenia, lactic acidosis.
AbacavirSevere hypersensitivity reaction; requires HLA-B*5701 screening if possible.
TenofovirRenal toxicity, decreased bone mineral density.
NevirapineHepatotoxicity, severe Stevens-Johnson syndrome rash.
Lopinavir/ritonavirMetabolic syndrome, diarrhea, lipodystrophy.

Special Clinical Situations

  • Tuberculosis Co-Infection: Rifampicin potently induces CYP450 enzymes, reducing antiretroviral levels.
  • Dolutegravir dosing must be doubled to twice daily during tuberculosis treatment.
  • Lopinavir/ritonavir requires super-boosting by increasing the ritonavir ratio to 1:1.
  • Immune Reconstitution Inflammatory Syndrome: Paradoxical worsening of pre-existing infections post-initiation requires continuing antiretroviral therapy and treating the underlying infection.